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Benjamin Reinking

Clinical Associate Professor,  Pediatrics

Contact Information

Phone: +1 319 356 2845
Email: benjamin-reinking@uiowa.edu


BA, University of Northern Iowa
MD, Medicine, University of Iowa
Residency, Pediatrics, University of Arizona
Fellowship, Pediatric Cardiology, University of Iowa
Training, Echocardiography, Lucille Packard Children's Hospital


Primary: Pediatrics

Centers and Program Affiliations

Research Interests

echocardiography, fetal echocardiography, hypoplastic left heart syndrome

MeSH Terms from Publications

Research Summary

Clinical Interests:
1) Director, Pediatric Cardiology Graphics Laboratory
2) Transesophageal, transthoracic, and fetal echocardiography
3) The diagnosis and treatment of fetal congenital heart disease
4) The noninvasive assessment of cardiac anatomy and function
5) Hypoplastic left heart syndrome

Research Interests: 
The use of high frequency ultrasound to evaluate cardiac anatomy and function in a basic science setting.

Recent Publications

Show publications
  1. Agoudemos, M, Reinking, B, Koppenhafer, S, Segar, J, Scholz, T. Programming of adult cardiovascular disease following exposure to late-gestation hyperglycemia. Neonatology 100(2):198-205, 2011. [PubMed]
  2. Bartlett, H, Escalera, R, Patel, S, Wedemeyer, E, Volk, K, Lohr, J, Reinking, B. Echocardiographic assessment of cardiac morphology and function in Xenopus. Comp Med 60(2):107-13, 2010. [PubMed]
  3. Wang Q, Lin J, Reinking BE, Feng H, Chan F, Lin C, Jin J, Gistafson-Wagner EA, Scholz TD, Yang B, Lin J. Essential roles of an intercalculated disc protein, xXinB, in postnatal heart growth and survival. Circulation Research 106(9):1468-78, 2010.
  4. Divekar, A, Morriss, J, Reinking, B, Bartlett, H. Transcatheter diagnosis and intervention for iatrogenic right-to-left shunts decades after surgical repair of partial anomalous pulmonary veins and an atrial septal defect. Pediatr Cardiol 30(8):1150-3, 2009. [PubMed]
  5. Reinking, B, Wedemeyer, E, Weiss, R, Segar, J, Scholz, T. Cardiomyopathy in offspring of diabetic rats is associated with activation of the MAPK and apoptotic pathways. Cardiovasc Diabetol 8:43, 2009. [PubMed]